SIDS: Serotonin neurons contribute to fail-safe mechanism that ensures recovery from interrupted breathing

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Research in mice adds to evidence that dips in the activity of serotonin neurons may increase SIDS risk. The study reveals the activity of serotonin-producing neurons in the infant mouse brain contributes to a fail-safe switch that allows recovery from interrupted breathing. If replicated in human studies, findings could pave the way to screening for SIDS risk, new therapies.

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